Prenatal testosterone exposure induces insulin resistance, uterine oxidative stress and pro-inflammatory status in rats
Artículo
Acceso abierto
Artículo
CONICET Digital
Prenatal androgen excess is considered one of the main causes of the development of polycystic ovary syndrome. In this study, we investigated the effect of prenatal hyperandrogenization (PH) on the physiology of the adul...
Idioma eng
Acceso abiertoRuta libre sin proxy. Acceso recomendado cuando no hay suscripción activa.
Open Access