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A Novel Player in Atherosclerotic Diseases in Renal Patients: Tumor Necrosis Factor-like Weak Inducer of Apoptosis (TWEAK)

Özkan GÜNGÖR et al · Turkish Society of Nephrology · 2019

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Cardiovascular disease is common in patients with chronic kidney disease. Atherosclerosis has a very important role in pathogenesis of cardiovascular diseases. Tumor Necrosis factor- Like Weak Inducer of Apoptosis (TWEAK) is a glycoprotein that belongs to the TNF superfamily. TWEAK plays important roles in cellular proliferation, growth, migration, osteoclastogenesis, angiogenesis and apoptosis. In the recent years, TWEAK has been suggested to have a role in atherosclerosis. While it is known that TWEAK levels are decreased in patient with renal failure, its association with atherosclerosis is not well-defi ned. This review focuses on the association of TWEAK with atherosclerosis in patients with renal patients.

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APA 7

al, Ö. G. E. (2019). A Novel Player in Atherosclerotic Diseases in Renal Patients: Tumor Necrosis Factor-like Weak Inducer of Apoptosis (TWEAK). https://turkjnephrol.org/index.php/pub/article/view/896

MLA

al, Özkan GÜNGÖR et. "A Novel Player in Atherosclerotic Diseases in Renal Patients: Tumor Necrosis Factor-like Weak Inducer of Apoptosis (TWEAK)." 2019. https://turkjnephrol.org/index.php/pub/article/view/896.

Chicago

al, Özkan GÜNGÖR et. 2019. "A Novel Player in Atherosclerotic Diseases in Renal Patients: Tumor Necrosis Factor-like Weak Inducer of Apoptosis (TWEAK).". https://turkjnephrol.org/index.php/pub/article/view/896.

Harvard

al, Ö. G. E. 2019, A Novel Player in Atherosclerotic Diseases in Renal Patients: Tumor Necrosis Factor-like Weak Inducer of Apoptosis (TWEAK), Turkish Society of Nephrology, available at: https://turkjnephrol.org/index.php/pub/article/view/896 [Accessed 29 Jun. 2026].

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Título
A Novel Player in Atherosclerotic Diseases in Renal Patients: Tumor Necrosis Factor-like Weak Inducer of Apoptosis (TWEAK)
Autor / colaboradores
Özkan GÜNGÖR et al
Editorial
Turkish Society of Nephrology
Año de publicación
2019
ISSN
2667-4440
ISSN
2667-4440
Idioma
eng

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