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Artículo de revista

Vascular Calcifi cation in Chronic Kidney Disease

Kültigin TÜRKMEN et al · Turkish Society of Nephrology · 2019

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"ONE HUNDRED AND THIRTY-FIVE HOURS UNDER THE RUIN : A CASE WITH TRAUMATIC RHABDOMYOLYSIS IN GÖLCÜK EARTHQUAKE"

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Summary

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The most common cause of death in patients with end-stage renal disease is cardiovascular (CV) disease. Vascular calcifi cation (VC) is a CV risk factor among dialysis patients. Advanced age, dialysis adequacy, vitamin D therapy and calcium-containing phosphate binders are additional clinical risk factors associated with VC. However, VC is a dynamic and programmed process and cannot only be attributed to passive calcium phosphate deposition. Osteoprotegerin, receptor activator nuclear factor kB (RANK) and its ligand (RANKL), along with the monocyte colony stimulating factor (M-CSF) and the transcription factor core binding protein (Cbfa-1) play a pivotal role in the control of VC. In contrast, Fetuin-A, matrix Gla protein (MGP) and osteopontin (OPN) can control the inhibition of VC. VC can be diagnosed in clinical practice with multi-detector computed tomography (MDCT) and electron beam computed tomography (EBCT).

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APA 7

al, K. T. E. (2019). Vascular Calcifi cation in Chronic Kidney Disease. https://turkjnephrol.org/index.php/pub/article/view/701

MLA

al, Kültigin TÜRKMEN et. "Vascular Calcifi cation in Chronic Kidney Disease." 2019. https://turkjnephrol.org/index.php/pub/article/view/701.

Chicago

al, Kültigin TÜRKMEN et. 2019. "Vascular Calcifi cation in Chronic Kidney Disease.". https://turkjnephrol.org/index.php/pub/article/view/701.

Harvard

al, K. T. E. 2019, Vascular Calcifi cation in Chronic Kidney Disease, Turkish Society of Nephrology, available at: https://turkjnephrol.org/index.php/pub/article/view/701 [Accessed 10 Aug. 2026].

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Title
Vascular Calcifi cation in Chronic Kidney Disease
Author / contributors
Kültigin TÜRKMEN et al
Publisher
Turkish Society of Nephrology
Publication year
2019
ISSN
2667-4440
ISSN
2667-4440
Language
English

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